Muscle defense of muscle: causes, symptoms and treatment

Deviation from muscles, under the protection of the disappearance of muscle removal, is a model used to brighten the mechanisms behind muscle atrophy, where it is observed when the muscle capacity is removed early muscle mass loss of 7 days, and this rapid loss is due to a decline in musclephibian protein. Ubiquitin-rort teasome system is responsible for the decline of muscle fiber-like protein, but the size of the response due to the removal of the inner suspension of the characteristics of the protocol is significantly variable. There are two main types of muscle capacity, and it is as follows: 1. Acute deletion is the contrast of muscles and muscle mass is normal in the individual, and the early mark can occur in people with the disease in the form of T2 (T2) in magnetic resonance imaging. Acute muscle capacity can occur as a result of the following: the crop that causes pressure, or the displacement of the nerve vascular package. Nerve -breaking due to shock. 2. Subacute and chronic disorder in these types of less specific features can prevent muscle atrophy with an increase in T1 (T1) signal in magnetic resonance imaging, and another signal that looks like heterogeneous edema. Symptoms of the disappearance of muscle removal can show some signs of muscle revenue, the most prominent of which are: muscle weakness. Muscle atrophy. Muscle tillage and tremor. The causes and factors of the risk of muscle ability. Growth and DNA damage indicated 45 Alpha – Gadd45a is an important molecular broker for muscle caused by the removal of inner suspension. It occurs as follows: significantly increasing the removal of structural muscle capacity from the level of this protein in muscle fibers and skeleton. It changes the genetic expression of the structural muscles in a way that stimulates the cracks of protein, reduces protein production, reduces mitochondria and inhibited by structural signals, and eventually causes muscle fiber atrophy. The reason for raising protein leading to the disease is unknown. The complications of the disappearance of muscle removal The complications that occur in different periods differ as follows: 1. The complications of short -term complications are the most prominent complications that occur during a short period of infection with the disappearance of muscle cut. The following: Muscular atrophy that varies according to the type of muscle. Muscle fibroid loss. Loss of keton and individual trends. Changes in size, number and the direction of the components of the anxious sarcoplasmia system. A decrease in the number of mitochondria. An increase in blood flow to kapillêres in the beginning and then decreased. 2. Long -term complications are the most important complications that occur after a period of injury. A great loss in the capillary -related to muscle fibers and degenerative changes in muscle nerve tree strains. The appearance of newly composed muscle fibers. Diagnosis of muscle revenue there are three main ways to diagnose the removal of muscle revenue, which is as follows: 1. It is known that the physical examination is removed the muscle capacity in the patient by signs of muscle weakness, wastage and muscle spasms fed by the nerve, or the root in demand. Clinical evidence indicates the removal of muscle removal to damage the lower motor nerve cells that harden these muscles anywhere from the cell body level to the peripheral neurological ends. More accurate tests for motor nerve cells need to be performed to know the affected muscles. 2.. The muscle -electric scheme contains the characteristic features of the removal of the inwardness of the muscle -electric plan. The presence of capabilities for fibrillation, positive acute waves and increased input activity. These are all forms of abnormal automatic activity. The capabilities of fibrillation have two forms of a two -phase spike, and the form of a single phase acute wave, both of which are short -term discharge with a small capacity. In the muscle affected by the disappearance of inner suspension, there is an increasing number of acetylcholine receptors on the surface of the muscle membrane, causing the muscle fibers that are extracted from the highly sensitive inner suspension of any of the relative acetylcholine, and which can automatically be removed, leading to fibrillation, and the removal of the presence of the fibrillation is a sign of the fibrillation, and it is the removal of the fibrillation capacity in the suitable position. Muscle inner continuous. 3. The muscle biopsy can be seen as proof of the disappearance of muscle removal by examining a biopsy sample of affected muscles using standard textile chemistry techniques. Changes in muscle fibers are attributed to the disappearance of inner suspension to various factors, including acetylcholine deficiency, the lack of mechanical electrical activity of muscle fibers, and the absence of nutritional factors transferred from the nerve to muscles. Treatment of muscle revenue is the treatment of muscle capacity treated as follows: 1. The treatment in the hospital gets a calming and local anesthesia as the doctor uses fluoroscopy to lead a needle to the nerves that cause pain, and then a local anesthesia is injected to nerve anesthesia. Then the doctor heats the needle to destroy a portion of the nerve and prevents him from sending pain signals. 2. Turnapy needs the patient to follow restrictions to eat and drink after receiving treatment at home, and he will need a person to take it to the home, and he may also receive medicine to relieve. It can also be recommended by physical therapy if necessary. Prevention of the disappearance of muscle removal, there are no thoughtful ways to prevent muscle interaction.